Septate branching acute angle branching - aspergillus
Immunofluorescence.
Microscopy - Dichroic mirror (allows only slight)
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Background-Black
Acanthamoeba - optically greenish white - cell wall
calcofluorwhite stain.
INFLAMMATION
-Acute inflammation-
I) Vascular events - Ist Transient vasoconstriction
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Vasodilatation
Vascular Permeability Hallmark of acute inflammation.
Stasis (So that neutrophil can deviate to periphery).
Vascular Permeability
1) Endothelial gap - (m/c-mechanism)
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at the level of venules. except lungs - capillaries.
- Immediate/Transient response.
2) Direct endothelial injury - Immediate prolonged
mild endothelial injury - Sunburn/Radiation, hemostatic
delayed prolonged 2 types than eter
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Firstranker's choice - leucocyte mediated delayed prolonged
Endothelial gap
Endothelial
Contraction (dominant)
- active
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- immediate
Transient
Chemical mediators.
Histamine/Serotonin.
Cm + R
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Oxidative Phosphorylation
ATP production (short time, lanergy)
(active)
Polymeration of cytoskeletal Protein
II Cellular events - Intravascular
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1) Margination - Transient attachment of Neutrophils to cell wall require Selectin
P Selectin - Rolling only
E - Selectin - endothelial
L -
2) Rolling - By Pselectin.
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3) Adhesion - By Integrin - V-CAM.
I-CAM.
4) Pavementing. Overlayering of neutrophils
Cytokine activate both endothelium & Neutrophil express
Intravascular: ICAM-1
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PECAM-1
5) Diapedesis - Transmigration of leucocytes through endothelial gap ; PECAM-1- CD-31 (most P-adhesion molecule for diapedesis)
6) Chemotaxis - unidirectional movement, toward chemotactic stimuli.
?) Exogenous - Bacteria Toxin
?) Complements - C5a > C3a > C4a.
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most-potent anaphylatoxin - C5a.
iii) Cox pathway - PGE2a PGD2a (for neutrophil)
iv) Lipoxygenase Pathway - LTB4
Phagocytosis
Adhesion molecules
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1) Selectin - cell to cell interaction only.
2) Integrin - cell to cell & cell to matrix interaction.
3) Ig Surface molecule - PECAM
4) Mucin like protein - Heparan sulphate,
Leucocyte-Adhesion Defect
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Autosomal Recessive
Recurrent infection.
Neutrophilia [Leukocytosis]
LAD -I
LAD-II
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m/c Sialyl Lewis ligand chrous. FERMTT-13.
aggressive a/ Bombay Blood group gene mutation
B chain of B2 integrin immune deficiency
Bleeding like glanzmann
delayed-corp thrombasthenia
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separation of umbilical cord stump.
1) Phagocytosis
Eosinophils also phagocytic
Opsonins ECM protein (fibronectin) can also be aopsonin
Best-IgG > Igm.
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C3b.
Steps of Phagocytosis
I Recognition due to opsonization.
Scavenger Receptor - Fc IgG.
Attachment C3b
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Ca2+ GTP1
polymeration of actin
Filipod
II Engulfment
Phagosome
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Killing lysosomes
Lysosome-Respiratory
acid Hydrolases Phagolysosome. Burst Release of (granules)
Degradation
Killing
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O2 dependent
m/c; m-effective
NADPH
oxidase O2 (superoxide)
SOD
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H2O2-MPO
H2O2
Halide
MPO
System
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Cl-
HOCl-
most effective bactericidal
agent in Phagocytic cells
Eosinophilic peroxide + H2O2
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I- HOI most effective Parasiticial
(Hyporodide agent
O2 independent Killing
Lactoferrin.
Lysozyme.
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sugar Basic protein
Phagocytosis - Chronic Granulomatous disease
defective Respiratory burst PHOX gene mutation
membranous cytoplasmic
m/c type Autosomal Recessive
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X-linked recessive -less common.
Gp91phox P67 PHOX
Chediak Hegashi Syndrome-
Defect Chemotaxis << Phagocytosis
List gene mutation (Lysosomal Transferase)
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Neutrophil infection.
Platelets - Bleeding disorder.
Factor V, VIII, Fibrinogen.
PAF-4-
Schwann cells - Peripheral neuropathy
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Melanocytes Albinism:
Dark-purple granules. (Dohle bodies)
Damaged Rough endoplasmic Reticulum
Septicemia m/c Cause
MDS
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Chediak Hegashi Syndrome
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This download link is referred from the post: Medical PG Handwritten Notes 1st Year, 2nd Year, 3rd Year and 4th Year (Study Material)