Hearing
loss
Organic
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Nonorganic
Conductive
Sensory
neural
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Sensorineural
sensory
Neural
Peripheral
Central
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8th nerveAny disease process which interferes with the
conduction of sound to reach cochlea causes
conductive hearing loss
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Negative Rinne test, i.e. BC > AC.Weber lateralized to poorer ear
Normal absolute bone conduction
Low frequencies affected more
Audiometry shows bone conduction better than
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air conduction with air-bone gapLoss is not more than 60 dB.
Speech discrimination is good
Congenital
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Meatal atresiaFixation of stapes footplate
Fixation of malleus head
Ossicular discontinuity
Congenital cholesteatoma
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Any obstruction in the ear canal, e.g. wax, foreign
body, furuncle, acute inflammatory swelling, benign
or malignant tumour or atresia of canal
Perforation of tympanic membrane, traumatic or
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infectiveFluid in the middle ear, e.g. acute otitis media, serous
otitis media or haemotympanum
Mass in middle ear, e.g. benign or malignant tumour
Disruption of ossicles, e.g. trauma to ossicular chain,
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chronic suppurative otitis media, cholesteatomaFixation of ossicles, e.g. otosclerosis,
tympanosclerosis, adhesive otitis media
Eustachian tube blockage, e.g. retracted tympanic
membrane, serous otitis media
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Sensorineural hearing loss (SNHL) results from lesions
of the cochlea, VIIIth nerve or central auditory
pathways
A positive Rinne test, i.e. AC > BC
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Weber lateralized to better ear.Bone conduction reduced on Schwabach and absolute
bone conduction tests.
More often involving high frequencies.
No gap between air and bone conduction curve on
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audiometryLoss may exceed 60 dB.
Speech discrimination is poor. 8. There is difficulty in
hearing in the presence of noise
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PRENATAL CAUSESInfant factors
Scheibe dysplasia-Dysplasia is seen in the cochlea
and saccule
Alexander dysplasia-It affects only the basal turn of
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membranous cochleaBing?Siebenmann dysplasia. There is complete
absence of membranous labyrinth.
Michel aplasia. There is complete absence of bony
and membranous labyrinth.
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Mondini dysplasia. Only basal coil is present orcochlea is 1.5 turns
Enlarged vestibular aqueduct.
Semicircular canal malformations
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Waardenburg syndrome-White forelock
Heterochromia iridis ,Vitiligo ,Dystopia
canthorum
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Usher syndrome-Retinitis pigmentosa ,Nightblindness
Pendred syndrome-Goitre ,Perchlorate discharge
test shows defect in organic binding of iodine
Alport syndrome-Hereditary progressive
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glomerulonephritis ,Corneal dystrophyInfections during pregnancy. -toxoplasmosis,
rubella, cytomegaloviruses, herpes type 1 and 2
and syphilis
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Drugs during pregnancy. Streptomycin,gentamicin, tobramycin, amikacin, quinine or
chloroquine
Radiation to mother in the first trimester
Nutritional deficiency, diabetes, toxaemia and
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thyroid deficiencyAnoxia
Prematurity and low birth weight
Birth injuries
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Neonatal jaundiceNeonatal meningitis
Sepsis
Time spent in neonatal ICU
Ototoxic drugs
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Genetic-Deafness may occur alone as in familial
progressive sensorineural deafness or in
association with certain syndromes, e.g. Alport,
Klippel-Feil, Hurler
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NongeneticViral infections (measles, mumps, varicella,
influenza), meningitis and encephalitis.
Secretory otitis media.
Ototoxic drugs.
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Trauma, e.g. fractures of temporal bone, middle earsurgery or perilymph leak.
Noise-induced deafness
Infections of labyrinth--viral, bacterial or spirochaetal
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Trauma to labyrinth or VIIIth nerve, e.g. fractures oftemporal bone or concussion of the labyrinth or the ear
surgery
Noise-induced hearing loss
Ototoxic drugs
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PresbycusisM?ni?re's disease
Acoustic neuroma
Sudden hearing loss
Familial progressive SNHL
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Systemic disorders, e.g. diabetes, hypothyroidism, kidneydisease, autoimmune disorders, multiple sclerosis, blood
dyscrasias.
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INFLAMMATIONS OFLABYRINTH
Viral labyrinthitis
Measles, mumps and cytomegaloviruses
Viruses usually reach the inner ear by blood stream
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BacterialBacterial infections reach labyrinth through the middle
ear (tympanogenic) or through CSF (meningogenic)
Syphilitic
Sensorineural hearing loss is caused both by
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congenital and acquired syphilisM?ni?re's syndrome
Hennebert's sign. A positive fistula sign in the
absence of a fistula. This is due to fibrous
adhesions between the stapes footplate and the
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membranous labyrinthTullio phenomenon
It is a genetic disorder in which there is
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progressive degeneration of the cochleastarting in late childhood or early adult life.
Hearing loss is bilateral with flat or basin-
shaped audiogram but an excellent speech
discrimination
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Various drugs and chemicals can damage the
inner ear and cause sensorineural hearing loss,
tinnitus and sometimes vertigo
Eg: Aminoglycoside, Diuretics,Salicylates
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NOISE TRAUMA
Acoustic trauma
Permanent damage to hearing can be caused
by a single brief exposure to very intense sound
without this being preceded by a temporary
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threshold shiftexplosion, gun fire or a powerful cracker and
may reach or cross 140 dB
Noise-induced hearing loss
chronic exposure to less intense sounds
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Temporary threshold shift (TTS). Thehearing is impaired immediately after
exposure to noise but recovers after an
interval of a few minutes to a few hours
even up to 2 week
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Permanent threshold shift (PTS). Thehearing impairment is permanent and does
not recover at all.
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Immune-mediated inner ear disease causesprogressive bilateral sensorineural hearing loss.
It occurs between 40 and 50 years with equal
incidence in both sexes.
Nearly 50% of patients also experience vestibular
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symptoms like disequilibrium, motion intolerance,positional or episodic vertigo
15% of patients have evidence of other
autoimmune disorder such as ulcerative colitis,
systemic lupus, rheumatoid arthritis or multiple
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sclerosisNo Organic lesion
Malingering or Psychogenic
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